adrenomyeloneuropathy magnesium glutathione Silencing of Abcd1 and Abcd2 genes sensitizes astrocytes for inflammation: implication for X-adrenoleukodystrophy* Glutathione imbalance in patients with

Author contributions NT performed most of the experiments in the cell lines, coordinated the collection of patient samples, processed all samples, conducted experiments, analyzed data, and wrote most of the manuscript

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Compared to controls, SLC25A39-deficient A549 and H1299 cells exhibited significantly elevated reactive oxygen species (ROS) levels, as indicated by increased green fluorescence intensity of DCF-DA staining (Fig