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and (3) to determine the involvement of the nicotinamide adenine dinucleotide phosphate (NADPH) oxidase (NOX) in the AFB1-mediated damage and its possible modulation by the antioxidant

This creates a mini-fasted state that partially preserves bioavailability

Exposed integrin 51 receptors in 30-m-thick sections were twice as frequent in unconstrained tissue as in constrained (Table 1)