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A higher turnover of dendritic spines contributes to increased synaptic plasticity, thus facilitating experience-dependent learning

GLP-1R signaling enhances glucose-dependent insulin secretion, suppresses inappropriately high glucagon after meals in many settings, slows gastric emptying, and engages central satiety circuitry (brainstem/hypothalamic nuclei) to reduce energy intake

Using tandem mass spectrometry as previously described 11 , we measured plasma levels of PAA, PAGly, and PAGln

It is plausible that both routes operate in parallel, with spatial, temporal, and metabolite-specific dynamics shaping functional outcomes

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