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46 The data from these mice, similar to those subjected to a transient increase in gut permeability, suggest that under conditions of compromised gut permeability, there are adaptive immune components activated in response to the hyperactivated immune system, and that these immune compensatory mechanisms may help provide resistance to colitis

At the cellular level, AOD-9604 exerts its primary effects through a complex mechanism involving beta-3 adrenergic receptors located on the surface of adipocytes (fat cells)