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Based on the increase in cardiolipin biosynthesis, we hypothesize that luseogliflozin facilitates the maintenance of mitochondrial ATP production in response to I/R injury-induced tissue damage and oxidative stress

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found that ACSL4 gene knockout could attenuate ischemic brain injury while over-expression aggravated IS (Doll and Conrad, 2017

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How metabolic heterogeneity shapes mitochondrial adaptation and differential therapeutic response Metabolic heterogeneity within CRC reflects more than spatial differences in energy pathways