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Description

An abnormal sulphydryl compound in urine

Mdivi-1 inhibits excessive mitochondrial fission by targeting Drp1, improving mitochondrial function in AD and HD models

Simultaneously, the second oxidative stress mechanism revolves around the perturbation of redox signaling pathways

The percent of BrdU+ cells co-expressing both DCX and NeuN was similarly higher in the TAM chow-fed mice than vehicle chow-fed mice (35.15 2.17% veh chow vs 48.09 3.42% TAM chow