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To further explore the role of SNX2, we conducted all-atom molecular dynamics simulations, which revealed that the AH domain of the SNX1SNX2a complex resists membrane insertion, as the average AHmembrane distance does not decrease over the course of the simulation (Supplementary Fig

In the small intestine, pancreatic proteases partially degrade the R proteins, releasing B12 to bind with another protein called intrinsic factor

Acknowledgements This work was supported by Science and Engineering Research Board (SERB) project no: SB/YS/LS-194/2013

Immune modulation by complement receptor 3-dependent human monocyte TGF-1-transporting vesicles

After three to four freeze-thaw cycles, even a high-purity peptide will show aggregation upon reconstitution