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In the myocardium, oxidative stress caused by ischemia/reperfusion (I/R) injury triggers nuclear translocation of nuclear respiratory factor 1 (NRF1) and upregulation of PPARG coactivator 1 alpha (PGC-1), which increase TFAM transcription and contribute to mitochondrial biogenesis and repair, acting as a compensatory mechanism(92)

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47 were the first to demonstrate that endogenous NGF protein in the sciatic nerve of STZ-diabetic rats declines progressively with disease duration

Given that exercise and tDCS share common neural substrates, it is hypothesised that combining these techniques produces synergistic effects, yielding cognitive enhancement outcomes superior to those of single interventions

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