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pinnata likely induce apoptosis in osteosarcoma cells through a dual-pathway mechanism involving both death receptor activation and mitochondrial signaling

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Intriguingly, Sandyk et al

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129 Paroxetine has also been shown to inhibit the inflammatory response in UC by blocking the interaction between G protein-coupled receptor kinase 2 (GRK2) and prostaglandin E2 receptor 4 (EP4), thereby reducing cyclic adenosine monophosphate (cAMP) levels and regulating M2 macrophage polarization