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The synthesis and expression of GPx4 can be modulated through various mechanisms, including transcriptional regulation, translational control, post-translational modifications, and epigenetic alterations

Chemical shifts () are reported in parts per million (ppm)

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Mice lacking radixin, which links actin filaments to plasma-membrane proteins, develop conjugated hyperbilirubinemia associated to retrieval of Mrp2,122 and the same hols true for obstructive and estrogen-induced cholestasis, where a disturbed colocalization of Mrp2 and radixin is associated with Mrp2 endocytic internalization.123 Interestingly, the internalization of Mrp2 that occurs after hepatic ischemia-reperfusion is coincident with a virtual loss of radixin expression.124 Mrp2 retention in the apical membrane also requires interaction with the PDZ-domain protein, PDZK1.125 Since other canalicular transporters, such as Bsep, Mdr1 and Mdr2, do not contain obvious PDZ-interacting motifs, regulation by actin of the localization of these transporters must occur through a different subset of proteins