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However, in glioma multiforme, the high level of ANKDD1A methylation changes the metabolism of the cancer cells, thereby inhibiting the antitumor effect of ANKDD1A 59

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Williams et al., 1994) or inhibiting (Williams et al., 1994) the activity of the receptor, depending on the combination of subunits forming each NMDA receptor (Williams et al., 1994)

Studies were eligible if they reported published data in English relevant to endometriosis and at least one of the targeted cell-death modalities, with specific attention to Erastin or its analogs in in-vitro, ex-vivo, in-vivo, or human observational contexts