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Lack of hepcidin gene expression and severe tissue iron overload in upstream stimulatory factor 2 (USF2) knockout mice

Another recent screening identified that H3K9 demethylation-mediated upregulation of BCAT1 and subsequent BCAA metabolic reprogramming is able to enhance the capacity for sublethal epidermal growth factor receptor (EGFR) inhibitor (TKI) resistance by producing ROS scavengers in lung cancer, which is a cancer where EGFR mutations are commonly found (Wang et al., 2019b)

Fatty Acid Amide Hydrolase (FAAH)an upregulated serine hydrolase in tumorsregulates cancer proliferation, and its inhibitors exhibit anti-invasive/anti-metastatic effects, often synergizing with chemotherapeutics (261, 262)

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