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Primate segmental duplications: crucibles of evolution, diversity and disease

For example, while moderate ROS often drive tumorigenic processes by enhancing prosurvival signaling, high ROS levels can inhibit these same pathways, such as PI3K/AKT, leading to growth arrest or cell death

S., Gerecht S., Shieh H

This suggests T4 might play a role in disease pathogenesis or tissue repair

Correlation analysis of the p53 expression and enzymatic activities upon variations in cellular thiamine/ThDP levels indicates that p21 knockdown substitutes positive correlation of the p53 expression with the activity of 2-oxoglutarate dehydrogenase complex (OGDHC) for that with the activity of glutamate dehydrogenase (GDH)