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Review of genes and results for degenerative and immunological disorders pertaining to Nrf2 Nrf2 target genes Thimmulappa et al investigated Nrf2-regulated genes induced by the chemopreventive agent sulforaphane using oligonucleotide microarray

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Activation of PPAR drives elevated free fatty acid (FFA) oxidation and utilization, initially serving as a compensatory response to heightened metabolic demand in the diabetic heart and also shown in other porcine models of cardiorenal syndrome [10, 17]