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When administered at therapeutic levels, APAP is primarily metabolized via the glucuronidation pathway, producing non-toxic metabolites that the liver and kidneys can further process (Chiew and Isbister 2023)

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Lp(a) is the major carrier in plasma of oxPLs that are detected by E06 immunoreactivity (249) and these oxPLs associate with Lp(a) in preference to native LDL particles in human plasma (250)