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While the precise mechanisms linking these metabolic alterations to fibrotic remodeling remain to be fully elucidated, current studies strongly support amino acid metabolism as a central regulatory axis in fibrosis initiation and progression

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Enzymes such as gst-4 and glod-4 function to prevent reactive oxidants such as glyoxal and methylglyoxal from nonenzymatically binding to proteins forming more AGEs 32,35

L-carnitine exerts hepatoprotective effects primarily through mitochondrial mechanisms

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