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Given the central role of DCs in the immunological homeostasis of the airway mucosa, we will highlight the effects of tobacco smoke on pulmonary DC biology, and propose mechanisms through which tobacco smoke-exposed airway DCs could trigger allergic responses

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One of the primary pathways leading to caspase activation involves the death receptor pathway, where receptors such as Fas/CD95, following stimulation by FasL, recruit adapter proteins like FADD to initiate the activation of procaspase-8 [160]

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