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Prolonged cigarette smoke exposure alters mitochondrial structure and function in airway epithelial cells Respiratory Research Springer Nature Link Cigarette smoke increases susceptibility to infection in lung epithelial cells by upregulating caveolin dependent endocytosis PLOS One Frontiers Mitochondrial quality control: a pathophysiological mechanism and potential therapeutic target for chronic obstructive pulmonary disease Inflammatory mechanisms in COPD. Cigarette smoke (and other irritants) Download Scientific Diagram Cigarette smoke can reprogram cells in your airways, causing COPD to hang on after smoking ends

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