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The majority of cancer-associated p53 mutations are missense-mutations (73%), resulting in more than 2,000 different mutant proteins observed in cancer so far, followed by frameshifts (9%) and nonsense mutations (8%), which result in premature stop codons that produce truncated and non-functional proteins (Hainaut and Pfeifer Citation2016

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A definitive or final diagnosis cannot be made based on clinical features alone