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Impairment of the electron transport chain (ETC) is one possible mechanism for increased mitochondrial ROS production via reverse electron transport to ETC complex 1 [56, 57], which could also explain why mitochondrial transmembrane potential was disturbed in CSE-treated ARPE-19 cells

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The Russell Standard proposes that biochemical verification of abstinence should occur at 6 or 12 months following the quit date [24]

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