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Levels of p-ERK, p-p38 and p-JNK proteins in bronchial cells and lung tissue increased significantly in the smoking group, indicating that the MAPK-signaling pathway is involved in smoking-induced abnormal epithelial cell differentiation

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Importantly, given that the discovery GWAS ignored time to cessation in the former smokers group, the phenotype can be conceivably more heterogeneous, making extrapolation of causality from these genetic-associations seemingly complicated

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