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Tissue resident macrophages are long-lived cells and thus rely on fatty acid oxidation and oxidative phosphorylation for both redox balance and prolonged survival (3840)

The mechanism responsible for the inhibitory effect of high GSH concentrations on the rate of H 2 O 2 formation is unclear, but it may involve a competitive inhibition of CSH binding to the SOD1 active-site by GSH 65

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