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Interestingly, iron was found to alter the balance between M1/M2 macrophage polarization, leading to macrophage-driven inflammation and fibrogenesis in nonalcoholic fatty liver disease (NAFLD) [64]

Induction of these chaperones represents an adaptive stress response that protects cardiomyocytes by stabilizing the contractile apparatus and preserving sarcomeric organization during ischemic injury

On the other hand, the p75 NTR receptor has been reported to either induce or prevent apoptosis upon oxidative insult in both in vivo and in vitro models

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Knock out of ALCAT-1, an enzyme that promotes cardiolipin peroxidation protected against onset of cellular senescence and preserved mitochondrial function by reducing sensitivity to lipid oxidation