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Of important note, vitamin D may also be able to reduce excitotoxicity through its action on L-type calcium channels which are thought to modulate NMDA receptors (Hur et al., 2014), and to increase release of the anti-inflammatory cytokine, IL-10 (Selhub, 2002) to provide cysteine for the production of glutathione (Dalto and Matte, 2017), and this also allows glutamate to be used in the production of glutathione, thereby limiting glutamate availability for excitotoxicity (Wu et al., 2004)

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doi: 10.3389/fvets.2022.956814