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SPI1 inhibits oxidative stress-induced NLRP3 activation and subsequent cell pyroptosis and attenuates pyroptosis-induced cardiac fibroblast activation and ECM synthesis by promoting the transcription of SIRT5 and mediating the lysine demalonylation of GSTP1

L., Selvarajah, D., Emery, C

In summary, we propose an active-site loop-dependent conformational switch that parks the apoprotein of class II Grx in an enzymatically inactive state when no ironsulfur clusters are present