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PMCID: PMC5951015 Li H, Liu X, Zhu Y, Liu Y, Wang Y (2015) Magnolol derivative 002C3 protects brain against ischemia-reperfusion injury via inhibiting apoptosis and autophagy

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The mechanisms underlying these effects likely involve glutathione's role in reducing oxidative stress in metabolic tissues, supporting mitochondrial function and energy production, and modulating inflammatory pathways that affect metabolic regulation

A quick look at the chart indicates a huge separation between the ME/CFS patients (in red) and the healthy controls (in blue)

DBD involves the construction of a comprehensive protective network that spans oxidative stressmetabolic stressstructural homeostasisaging regulation through the systematic regulation of the antioxidant pathway (SODSOD2GSH-PxMDA), the restoration of ECM and metabolic homeostasis (MMP1 and PPAR), and the blockade of the senescence fate program (P21)