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However, as knowledge of oxidation/reduction (redox)-dependent signaling and molecular mechanisms of antioxidant therapies has increased, many are now questioning the traditional view of oxidative stress as a pro-oxidant/anti-oxidant imbalance and instead are focusing on oxidative stress as a disruption of redox signaling and control (156)

2024-BS-074), the Basic Scientific Research Fund of Northeastern University of China (N25LPY044)

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The BP class of GO annotations included the process of starch catabolism, response to salicylic acid and jasmonic acid, protein import into peroxisome membrane, and maintenance of protein location in cell