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This condition is a major contributor to physical decline, diminished quality of life, and reduced survival in patients with advanced malignancies [1,2,3]

Effect of ginsenoside Rh1 on proliferation, apoptosis, and oxidative stress in vascular endothelial cells by regulation of the nuclear erythroid 2-related factor-2/heme oxygenase-1 signaling pathway

We first examined apoptosis in Raji cells by flow cytometry (Fig

Similarly, in colorectal cancer stem cells, p300-mediated H4K12la upregulates glutamate-cysteine ligase catalytic subunit (GCLC), thereby expanding the GSH pool and conferring ferroptosis resistance (116)