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As a result, surgical damage to the hypothalamus is a key contributor to the risk of postoperative weight gain[99], which, perhaps surprisingly, does not seem to be exacerbated by radiation-based adjuvant treatment [31]

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TB-500 binds to G-actin monomers, preventing premature polymerization into filaments

Others indicate that decreases in NADPH/NADP could influence the redox state of the GSH pool

The human brain is especially vulnerable to oxidative stress because it accounts for only 2% of body mass but consumes 20% of oxygen 14