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Description

The current evidence suggests that the peptide exerts its activity through interactions with amylin receptors and related receptor complexes

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This difference stems from the essential differences in metabolic characteristics and signaling pathways: the redox imbalance of the GCB subtype makes it vulnerable to ferroptosis, while the high expression of BCL-2 in the ABC subtype gives it an anti-apoptotic survival advantage

Recently, proteolysis-targeting chimeras (PROTACs) that enhance the degradation of disease-related proteins of interest have been recognized as new therapeutics in drug discovery