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Cell death is a consequence of the decrease in ATP production which rapidly compromises ion (mainly due to halting Na + -K + pump) and acid-base homoeostasis

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The pharmacology in animal studies and early human studies supported this design: AOD-9604 stimulates lipolysis and inhibits lipogenesis through activation of beta-3 adrenergic receptors in adipose tissue, without producing the IGF-1 elevation or insulin resistance signal that exogenous GH produces