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Based on these data, Lombard proposed that ASD is a mitochondrial disease [66]

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Current therapies for hyperpigmentation typically target either ROS neutralization or AGEs inhibition, but often overlook the synergistic interaction between these pathways 3,20

However, an autoradiographic post-mortem study demonstrated no significant modifications in the expression of NMDA receptors in the hippocampal tissue from 4 males with autism [64]