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Indeed, lack of consistent CD-like enteropathy in humanized mice [61] supports the concept that the accelerated disruption of enterocytes secondary to the adaptive CD4 + T cell insult cannot fully explain CD pathogenesis, supporting the notion that an intrinsic defect of the stem cell compartment in subjects at risk of CD is a key element of CD enteropathy [60, 62]

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Given that one of the severely damaged regions following TBI is the cerebellar circuit, PQQ could stimulates neurite regeneration in a wound recovery model using cultured cerebellar granular neurons (CGNs) (Shanan et al., 2019)

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