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Through rigorous mechanistic, translational, and preclinical studies, this work aims to establish COPS5 as a target for a new therapy that will weaken the tumor and make it more susceptible to other known treatments, such as PARP inhibitors

Everett, S

doi: 10.1016/0014-5793(92)80288-r

30 A-D creatinine levels of all vehicle-treated C57BL/6 mice exceeded 2.0 mg/dL and showed strong morphologic damage and high serum urea levels whereas Fer-1-treated and, to a greater extent, SRS16-86-treated mice were protected from functional acute renal failure ( FIGS