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6b), resulting in an open pathway from the central pocket to the cytoplasm

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Subsequent studies demonstrated that exogenous administration of thymosin beta-4 promoted corneal wound healing, dermal repair, and cardiac tissue recovery in rodent models

Previous literature has shown that oxidative stress is a key factor in the progression and deterioration of autoimmune diseases, possibly by further inducing and expanding the expansion of pro-inflammatory Th17 cells, inhibiting the differentiation of anti-inflammatory Treg cells, and exacerbating autoimmune damage (3234)

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