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During hypoxia or under certain oncogenic conditions, -ketoglutarate could be converted directly to citrate (following a reversed TCA cycle), in order to generate the cytosolic acetyl-CoA when glucose-derived acetyl-CoA is insufficient [34]

Key features include: pH-sensitive linkers have demonstrated excellent drug accumulation efficiency and controllable release in preclinical studies, making them a critical direction in modern ADC development

Interestingly, such changes in mitochondrial NADH/NAD + redox are directly affected by glutamate oxaloacetate transaminase 2 (GOT2), a mitochondrial enzyme whose activity also contributes to tryptophan catabolism 69,70 and amino acid metabolism 71

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