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demonstrated that silencing PSAT1 reestablished sensitivity to erlotinib and enhanced its tumoricidal functions by activating the ROS-dependent JNK/c-Jun pathway [105]

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Consequently, the autonomic nervous system may not be forced to trigger compensatory tachycardia, potentially leading to a stabilization of heart rate and blood pressure

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Given LNPs favorable safety profiles and proven clinical success, their application in uterine fibroid therapy especially for gene-based or immunotherapeutic approaches holds potential