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The proposed mechanisms are as follows: counteracting the increased intracellular production of reactive oxygen species and the activation of pro-apoptotic caspase-3 enzyme, inhibiting the receptor of epidermal growth factor and the extracellular signal-regulated kinase (ERK) phosphorylation, inhibiting lipid peroxidation, reducing LDH activity, a potential activator of neuronal voltage-gated potassium channel subfamily Q (KCNQ)

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10.1016/0303-7207(95)03514-8 73 IwatsuboK.BravoC.UechiM.BaljinnyamE.NakamuraT.UmemuraM.et al (2012)

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