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S1C) indicating that the T94 2.61 I substitution is the responsible for the blue-shift, from 500 nm to 478 nm, observed in the other double mutants and in the triple mutant

PROBIOTIC STRAIN OF HUMAN ORIGIN

Although the mechanisms by which DUX4 exerts myotoxicity remain unclear, accumulating evidence has indicated that DUX4 induces oxidative stress, which plays an important role in FSHD pathogenesis (1, 2, 4, 14)

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Visomitin exhibited excellent renal protective effects in FA-, cisplatin-, and IR-induced models by reducing lipid peroxidation and mitochondrial ROS generation to ameliorate ferroptosis in kidneys in vivo and in vitro [189]