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Research suggests that it also contributes to cytoprotective processes by enhancing cellular resistance to chemical, mechanical, or ischemic stress

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Beyond breast cancer, female-specific vulnerabilities emerge in ovarian cancers associated with BRCA mutations, where disrupted NRF2-BRCA1 interactions compromise redox defenses and reveal unique therapeutic windows, reviewed in Li et al ( NRF2 activation in breast cancer enhances the expression of Rho and its downstream proteins like Focal adhesion kinase 1 (FAK) and Modulator of volume-regulated anion channel current 1 (MLC), while suppressing estrogen-related receptor (ERR1) ( in vitro (160, 161)

However, some polysaccharides exhibit high solubility and poor film-forming ability, leading to premature drug release and reduced colonic targeting