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Wild-type enzyme and K105X mutants tended to have infinite true k cat and K m values for extrapolated infinite substrate concentrations, suggesting that the enzymes are neither saturated by GSSCys nor GSH (Supplementary Fig

CTRP3 alleviates mitochondrial dysfunction and oxidative stress injury in pathological cardiac hypertrophy by activating UPRmt via the SIRT1/ATF5 axis

With the advancement of AI, there is growing interest in using machine learning and data analysis techniques to evaluate therapeutic outcomes

While simple dietary intervention is appealing, our understanding of the consequences of catabolite buildup remains incomplete

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