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Adding exogenous glutathione to a drug that already modulates oxidative stress raises a conceptual question: could the combined antioxidant load suppress reactive oxygen species (ROS) to a degree that interferes with beneficial ROS signaling

Finally, although the total brain lipid content may decrease with age, the enzymes responsible for incorporating PUFAs into membrane phospholipids remain active, ensuring the continued presence of the necessary substrates for the final execution step of ferroptosis (75)

Gmunder H, Droge W (1991) Differential effects of glutathione depletion on T cell subsets

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Cells 12 , 785 (2023)