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Therapeutic or experimental strategies targeting aging organize around three main axes: senolytics (selective elimination of senescent cells), senomorphics (SASP modulation without killing cells), and geroprotectors (reinforcement of anti-aging mechanisms like autophagy, mitophagy, telomerase activity)

The best way to ensure that your body remains healthy and you continue to feel good is to remove these toxins from your system before they have a chance to attack healthy cells

As mentioned above, there are more than 100 Akt substrates mainly including GLUT4, FOXO1, GSK3, mTORC1, SREBP-1c, TSC1/2, PRAS40, ABHD15, PDE3B

10.1016/j.bbamcr.2022.119417 Biochim

Similarly, the reduction in NCOA4 expression, a gene involved in the autophagic degradation of ferritin, known as ferritinophagy [36], further supports melatonins role in modulating iron metabolism to mitigate ferroptosis risk