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The regulatory landscape may change as additional safety and efficacy data become available, and healthcare providers must stay informed about evolving regulatory requirements and restrictions

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Here, we tested this hypothesis through various pharmacological approaches coupled to high-content kinetic cell death assays using super-resolution real-time live-cell microscopy and an in vivo xenograft model, revealing a potent and unrecognized coupling between IKE and DHAA that induces ferroptosis in tumor models otherwise resistant to IKE-induced ferroptosis

Specificity resides in the () Glutamyl portion of the substrate