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Rather, hypochloremia can promote sodium retention in several ways: Hypochloremia appears to stimulate renin secretion, thereby activating the renin/angiogensin/aldosterone axis

Excessive ROS can damage thyroid tissue by binding to membrane lipids and proteins, leading to the release of autoantigens

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Its proposed mechanism is unusual: Selank appears to modulate the expression of GABA-A receptor genes without binding the benzodiazepine site , which is offered as an explanation for anxiolytic activity that comes without sedation, tolerance, dependence or withdrawal [3]