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Adalimumab prevents barrier dysfunction and antagonizes distinct effects of TNF-alpha on tight junction proteins and signaling pathways in intestinal epithelial cells

Recent studies have demonstrated that EMPA exerts direct immunomodulatory effects, particularly through mechanisms that link metabolic regulation to inflammatory signaling

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Recently, it was demonstrated that in a high-glucose (HG) environment, the tumor suppressor gene BAP1 partially deubiquitinates H2Aub on the SLC7A11 promoter, thereby suppressing transporter function, limiting cysteine uptake, and increasing ferroptosis sensitivity [42]