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[25] Within the field of nutrition, the terms are often used interchangeably

Since GSTO1 has been known to cause de-glutathionylation of -actin impeding its polymerization into filamentous actin 11 , such a downregulation of molecules involved in cytoskeleton organization and adhesion is particularly interesting

Both mitochondrial Casp8 and Bid were negatively correlated with Gsta4 expression, further proving mitochondria as a relevant location for the intersection between 4-HNE and additional pathways, including the Fas-Casp8-Bid-axis

uncovered that aspirin regulated sphingolipid metabolism in dose-dependent manner, with low-dose aspirin (75 mg/day) increasing plasma S1P levels of volunteers, while high-dose (300 mg/day) decreasing S1P levels [53]

Organization of the human mitochondrial hydrogen sulfide oxidation pathway