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Regarding treatment strategies, it is suggested that the marked decrease in GPx1 expression may be the major mechanism of tumor sensitization to anthracyclines [93] and that GPx1 can partially inhibit doxorubicin-induced cell death-related signaling in breast cancer by interfering with the activation of the sphingomyelin-ceramide pathway [85]

This is just one of many examples from epigenetics that show how environmental influences can regulate our genes

While the treatment with Ru(II)-OH or NP2 slightly elevated the ROS levels inside the hypoxic cancer cells, the treatment with NP4 showed a strong enhancement of ROS

elegans can be extended by superoxide/catalase mimetics suggesting that oxidative stress is a major determinant of the rate of aging

10.1056/NEJMra1106180 N Engl J Med