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Product-quality risk is unquantified because grey-market vials are not independently characterized

In the setting of hyperlipidemia, the pathological effect of transsulfuration pathway on redox status is not that simple, because in normal physiological settings, the flux of homocysteine to GSH through the transsulfuration pathway can be compensatively enhanced in response to the oxidative stress in order to maintain the oxidant/antioxidant balance whereas antioxidant treatment elicit the opposite effect on transsulfuration pathway [8]

N-acetylcysteine inhibits platelet-monocyte conjugation in patients with type 2 diabetes with depleted intraplatelet glutathione: a randomised controlled trial

Curcumin-loaded porphyrin micelles (T-MP) were successfully prepared with a size smaller than 50 nm using a surfactant-stripping method

Theranostics 12 , 49354948 (2022)