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MDM2, murine double minute 2

The development of new technologies such as CRISPR/Cas9 and single-cell sequencing has opened up more possibilities for understanding the mechanisms of RCD and identifying new therapeutic targets

Their study demonstrated that this metabolic shift supports the hyper-proliferative and apoptosis-resistant phenotype by inhibiting AMPK and activating the AKT/mTOR signaling axis

Angiotensin-Converting Enzyme Inhibitors and AT1 Receptor Blockers Angiotensin II contributes to endothelial dysfunction, causing oxidative stress by NADPH oxidase activation (via the AT1-receptor)

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